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Free Radical Biol Med 39 : 345–354 Additional information Edited by B Zhivotovsky Rights and permissions About this article Cite this article Filomeni, G., Rotilio, G
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Conversely, deletion of HDAC3 increased expression of cathepsin B in, resulting in significant degradation of serine-threonine kinase 1 (RIP1), which interferes with TNF-α-mediated NF-κB activation and inflammatory response
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