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Pretreatment with individual ALC at 100 μM, and combined LA+ALC at 0.1 μM and 1 μM markedly inhibited the elevation of protein carbonyls

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This degradation, in turn, allows NF-κB activity to increase, resulting in increased transcription of pro-inflammatory genes, including TNF-α, IL-1β, IL-6, and inducible nitric oxide synthase (iNOS) (Morgan and Liu, 2011)

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