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However, in addition to the PCC, studies using positron emission tomography have consistently identified hypermetabolism at rest in brain regions comprising a cortico-striatal-thalamo-cortical circuit in OCD patients – most notably orbitofrontal cortex, (54–57) anterior cingulate cortex, (31, 57) caudate nuclei, (54, 55) putamen, (31) and thalamus (31) – suggesting that decreased GSH/Cr, and increased oxidative stress, may be present in some of these brain regions as well
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[80] reported that ferroptosis plays an important role in HCV replication
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