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Researchers observed that treatment was associated with reduced p16-positive cell populations and changes in physical function parameters in aged animals [1]
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The transcriptional regulatory activities of a TRIM on autophagy was first demonstrated for the PHD domain-containing TRIM28 (also known as KAP1, Table 1 ) ( Ulk1 , Becn1 , Atg12 ) and for proteins with mitophagy-specific functions ( Nix and Bnip3L )
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